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Senescence lncRNAs govern cell surface components: lncRNA-OIS1 transcriptionally elevates DPP4

  
@article{NCRI4934,
	author = {Rachel Munk and Kyoung Mi Kim and Myriam Gorospe and Kotb Abdelmohsen},
	title = {Senescence lncRNAs govern cell surface components: lncRNA-OIS1 transcriptionally elevates DPP4},
	journal = {Non-coding RNA Investigation},
	volume = {3},
	number = {0},
	year = {2019},
	keywords = {},
	abstract = {With advancing age, senescent cells accumulate in tissues and organs, accelerating aging and age-related disease (e.g., diabetes, neurodegeneration, and cancers) (1). Cellular senescence is triggered by sublethal stresses including telomere shortening (replicative senescence), damage to DNA or other molecules (premature senescence), and oncogenic activation (oncogene-induced senescence, OIS) (2-5).},
	issn = {2522-6673},	url = {https://ncri.amegroups.org/article/view/4934}
}